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The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction

The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction
2025-02-18
(Press-News.org) In your coverage, please use this URL to provide access to the freely available paper in PLOS Biology: https://plos.io/42Ly2Pr  

Article title: Capsid transfer of the retrotransposon Copia controls structural synaptic plasticity in Drosophila

Author countries: United States

Funding: This work was supported by NIH Grant R01NS112492 to TT. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.

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The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction

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[Press-News.org] The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction