(Press-News.org) A new study presented today at the International Liver CongressTM shows a novel pathway where activated natural killer (NK) cells expressing death ligands may excessively down-modulate the antiviral immune response in chronic HBV patients.1
Blocking the TNF-related apoptosis-inducing ligand (TRAIL) pathway partially reconstitutes HBV-specific T cells, suggesting that these cells are vulnerable to NK cell-mediated apoptosis through this death ligand pathway.
NK cells are one of the main effectors of the innate immune response that plays a key role in containing intracellular pathogen infections. However, it has been increasingly recognised that NK cells may also exert a pathogenic and negative regulatory role during chronic disease.
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References
1. Peppa, D. et al. Fratricide of HBV-specific CD8 T cells by NK cells mediated through the TRAIL pathway. Abstract presented at The International Liver CongressTM 2011.
Fratricide of HBV-specific CD8 T cells by NK cells mediated through the TRAIL pathway
Innovation in science
2011-04-02
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[Press-News.org] Fratricide of HBV-specific CD8 T cells by NK cells mediated through the TRAIL pathwayInnovation in science